Publicacións en colaboración con investigadores/as de Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas (9)

2024

  1. Copper Overload Increased Rat Striatal Levels of Both Dopamine and Its Main Metabolite Homovanillic Acid in Extracellular Fluid

    International Journal of Molecular Sciences, Vol. 25, Núm. 15

  2. Neddylation orchestrates the complex transcriptional and posttranscriptional program that drives Schwann cell myelination

    Science Advances, Vol. 10, Núm. 15

2019

  1. Copper Increases Brain Oxidative Stress and Enhances the Ability of 6-Hydroxydopamine to Cause Dopaminergic Degeneration in a Rat Model of Parkinson’s Disease

    Molecular Neurobiology, Vol. 56, Núm. 4, pp. 2845-2854

2017

  1. Copper increases the ability of 6-hydroxydopamine to generate oxidative stress and the ability of ascorbate and glutathione to potentiate this effect: potential implications in Parkinson's disease

    Journal of Neurochemistry, Vol. 141, Núm. 5, pp. 738-749

  2. Effects of Aluminium on Rat Brain Mitochondria Bioenergetics: an In vitro and In vivo Study

    Molecular Neurobiology, Vol. 54, Núm. 1, pp. 563-570

2016

  1. Mitochondrial angiotensin receptors in dopaminergic neurons. Role in cell protection and aging-related vulnerability to neurodegeneration

    Cell Death and Disease, Vol. 7, Núm. 10

2013

  1. A simple method for isolating rat brain mitochondria with high metabolic activity: Effects of EDTA and EGTA

    Journal of Neuroscience Methods, Vol. 213, Núm. 1, pp. 39-42

2011

  1. Antioxidant properties of dimethyl sulfoxide and its viability as a solvent in the evaluation of neuroprotective antioxidants

    Journal of Pharmacological and Toxicological Methods, Vol. 63, Núm. 2, pp. 209-215

2009

  1. Brain oxidative stress and selective behaviour of aluminium in specific areas of rat brain: Potential effects in a 6-OHDA-induced model of Parkinson's disease

    Journal of Neurochemistry, Vol. 109, Núm. 3, pp. 879-888