Publikationen, an denen er mitarbeitet RAMON SOTO OTERO (11)

2017

  1. Effects of Aluminium on Rat Brain Mitochondria Bioenergetics: an In vitro and In vivo Study

    Molecular Neurobiology, Vol. 54, Núm. 1, pp. 563-570

2013

  1. A simple method for isolating rat brain mitochondria with high metabolic activity: Effects of EDTA and EGTA

    Journal of Neuroscience Methods, Vol. 213, Núm. 1, pp. 39-42

2012

  1. 2-Benzazepine nitrones protect dopaminergic neurons against 6-hydroxydopamine-induced oxidative toxicity

    Archiv der Pharmazie, Vol. 345, Núm. 8, pp. 598-609

  2. Differential toxicity of 6-hydroxydopamine in SH-SY5Y human neuroblastoma cells and rat brain mitochondria: Protective role of catalase and superoxide dismutase

    Neurochemical Research, Vol. 37, Núm. 10, pp. 2150-2160

2009

  1. Brain oxidative stress and selective behaviour of aluminium in specific areas of rat brain: Potential effects in a 6-OHDA-induced model of Parkinson's disease

    Journal of Neurochemistry, Vol. 109, Núm. 3, pp. 879-888

2008

  1. Inhibition of 6-hydroxydopamine-induced oxidative damage by 4,5-dihydro-3H-2-benzazepine N-oxides

    Biochemical Pharmacology, Vol. 75, Núm. 7, pp. 1526-1537

2007

  1. Analysis of brain regional distribution of aluminium in rats via oral and intraperitoneal administration

    Journal of Trace Elements in Medicine and Biology, Vol. 21, Núm. SUPPL. 1, pp. 31-34

  2. Angiotensin type-1-receptor antagonists reduce 6-hydroxydopamine toxicity for dopaminergic neurons

    Neurobiology of Aging, Vol. 28, Núm. 4, pp. 555-567

  3. Time-course of brain oxidative damage caused by intrastriatal administration of 6-hydroxydopamine in a rat model of Parkinson's disease

    Neurochemical Research, Vol. 32, Núm. 1, pp. 99-105

2006

  1. Study on the ability of 1,2,3/4-tetrahydropapaveroline to cause oxidative stress: Mechanisms and potential implications in relation to Parkinson's disease

    Journal of Biochemical and Molecular Toxicology, Vol. 20, Núm. 5, pp. 209-220

2004

  1. Autoxidation and MAO-mediated metabolism of dopamine as a potential cause of oxidative stress: Role of ferrous and ferric ions

    Neurochemistry International, Vol. 45, Núm. 1, pp. 103-116