Publicacións en colaboración con investigadores/as de Universidad Complutense de Madrid (29)

2013

  1. Importance of cerebral artery recanalization in patients with stroke with and without neurological improvement after intravenous thrombolysis

    Stroke, Vol. 44, Núm. 9, pp. 2513-2518

  2. Neuronal excitotoxicity after carotid angioplasty and stent placement procedures

    Radiology, Vol. 268, Núm. 2, pp. 515-520

2011

  1. The natural tissue plasminogen activator inhibitor neuroserpin and acute ischaemic stroke outcome

    Thrombosis and Haemostasis, Vol. 105, Núm. 3, pp. 421-429

2008

  1. Delayed post-ischemic administration of CDP-choline increases EAAT2 association to lipid rafts and affords neuroprotection in experimental stroke

    Neurobiology of Disease, Vol. 29, Núm. 1, pp. 123-131

2007

  1. A chronic treatment with CDP-choline improves functional recovery and increases neuronal plasticity after experimental stroke

    Neurobiology of Disease, Vol. 26, Núm. 1, pp. 105-111

  2. Increased body iron stores are associated with poor outcome after thrombolytic treatment in acute stroke

    Stroke, Vol. 38, Núm. 1, pp. 90-95

  3. Statin treatment withdrawal in ischemic stroke: A controlled randomized study

    Neurology, Vol. 69, Núm. 9, pp. 904-910

  4. The increase of circulating endothelial progenitor cells after acute ischemic stroke is associated with good outcome

    Stroke, Vol. 38, Núm. 10, pp. 2759-2764

2006

  1. A polymorphism in the EAAT2 promoter is associated with higher glutamate concentrations and higher frequency of progressing stroke

    Journal of Experimental Medicine, Vol. 203, Núm. 3, pp. 711-717

  2. High blood pressure and inflammation are associated with poor prognosis in lacunar infarctions

    Cerebrovascular Diseases, Vol. 22, Núm. 2-3, pp. 123-129

  3. Hyperthermia is a surrogate marker of inflammationmediated cause of brain damage in acute ischaemic stroke

    Journal of Internal Medicine, Vol. 260, Núm. 4, pp. 343-349

  4. Ischemic preconditioning: A novel target for neuroprotective therapy

    Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 38-47

  5. Micro- and macroalbuminuria predict hemorrhagic transformation in acute ischemic stroke

    Neurology, Vol. 67, Núm. 7, pp. 1172-1177

  6. Neurorepair versus neuroprotection in stroke

    Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 54-63

  7. New-onset hypertension and inflammatory response/poor outcome in acute ischemic stroke

    Neurology, Vol. 67, Núm. 11, pp. 1973-1978

  8. Rosiglitazone and 15-deoxy-Δ12,14-prostaglandin J 2 cause potent neuroprotection after experimental stroke through noncompletely overlapping mechanisms

    Journal of Cerebral Blood Flow and Metabolism, Vol. 26, Núm. 2, pp. 218-229

  9. Targets of cytoprotection in acute ischemic stroke: Present and future

    Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 1-8

2005

  1. Increased plasma levels of 15-deoxy Δ prostaglandin J2 are associated with good outcome in acute atherothrombotic ischemic stroke

    Stroke, Vol. 36, Núm. 6, pp. 1189-1194

  2. Inhibition of iNOS activity by 1400W decreases glutamate release and ameliorates stroke outcome after experimental ischemia

    Neurobiology of Disease, Vol. 18, Núm. 2, pp. 375-384

  3. Neuroprotection afforded by prior citicoline administration in experimental brain ischemia: Effects on glutamate transport

    Neurobiology of Disease, Vol. 18, Núm. 2, pp. 336-345