Fisioloxía e farmacoloxía das enfermedades crónicas
FIFAEC
Harvard Medical School
Boston, Estados UnidosPublicacións en colaboración con investigadores/as de Harvard Medical School (8)
2008
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Cyclin-dependent kinase antagonizes promyelocytic leukemia zinc-finger through phosphorylation
Oncogene, Vol. 27, Núm. 27, pp. 3789-3796
2001
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Binding of SV40 large T antigen to the retinoblastoma susceptibility gene product and related proteins.
Methods in molecular biology (Clifton, N.J.), Vol. 165, pp. 213-218
1998
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The J domain of simian virus 40 large T antigen is required to functionally inactivate RB family proteins
Molecular and Cellular Biology, Vol. 18, Núm. 3, pp. 1408-1415
1997
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DnaJ/hsp40 chaperone domain of SV40 large T antigen promotes efficient viral DNA replication
Genes and Development, Vol. 11, Núm. 9, pp. 1098-1110
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Inactivation of pRB-related proteins p130 and p107 mediated by the J domain of simian virus 40 large T antigen
Molecular and Cellular Biology, Vol. 17, Núm. 9, pp. 4979-4990
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Ras signalling linked to the cell-cycle machinery by the retinoblastoma protein
Nature, Vol. 386, Núm. 6621, pp. 177-181
1996
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Simian virus 40 large T antigen alters the phosphorylation state of the RB-related proteins p130 and p107
Journal of Virology, Vol. 70, Núm. 5, pp. 2781-2788
1995
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Role of pRb-related proteins in simian virus 40 large-T-antigen-mediated transformation
Molecular and Cellular Biology, Vol. 15, Núm. 10, pp. 5800-5810