Publicacións (27) Publicacións de MARIA CLARA ALVAREZ VILLAMARIN Ver datos de investigación referenciados.

filter_list Endocrinology

2025

  1. RET signalling in the pituitary: a double-edged sword for differentiation, apoptosis and therapeutic strategies in acromegaly

    Endocrine-Related Cancer, Vol. 32, Núm. 3

2024

  1. Integrative clinical, hormonal, and molecular data associate with invasiveness in acromegaly: REMAH study

    European Journal of Endocrinology, Vol. 190, Núm. 6, pp. 421-433

2021

  1. Antagonists of growth hormone-releasing hormone inhibit the growth of pituitary adenoma cells by hampering oncogenic pathways and promoting apoptotic signaling

    Cancers, Vol. 13, Núm. 16

  2. RET signalling provides tumorigenic mechanism and tissue specificity for AIP-related somatotrophinomas

    Oncogene, Vol. 40, Núm. 45, pp. 6354-6368

2020

  1. GFRα 1-2-3-4 co-receptors for RET Are co-expressed in Pituitary Stem Cells but Individually Retained in Some Adenopituitary Cells

    Frontiers in Endocrinology, Vol. 11

  2. Why Should Growth Hormone (GH) Be Considered a Promising Therapeutic Agent for Arteriogenesis? Insights from the GHAS Trial

    Cells, Vol. 9, Núm. 4

2019

  1. Unmasking a new prognostic marker and therapeutic target from the GDNF-RET/PIT1/p14ARF/p53 pathway in acromegaly

    EBioMedicine, Vol. 43, pp. 537-552

2017

  1. The syndrome of central hypothyroidism and macroorchidism: IGSF1 controls TRHR and FSHB expression by differential modulation of pituitary TGFβ and Activin pathways

    Scientific Reports, Vol. 7

2016

  1. El Registro Molecular de Adenomas Hipofisarios (REMAH): una apuesta de futuro de la Endocrinología española por la medicina individualizada y la investigación traslacional

    Endocrinología y nutrición: órgano de la Sociedad Española de Endocrinología y Nutrición, Vol. 63, Núm. 6, pp. 274-284

2015

  1. Pituitary Cell Turnover: From Adult Stem Cell Recruitment through Differentiation to Death

    Neuroendocrinology, Vol. 101, Núm. 3, pp. 175-192

2014

  1. Somatotropinomas, but not nonfunctioning pituitary adenomas, maintain a functional apoptotic RET/Pit1/ARF/p53 pathway that is blocked by excess GDNF

    Endocrinology, Vol. 155, Núm. 11, pp. 4329-4340

2013

  1. The orexigenic effect of orexin-A revisited: Dependence of an intact growth hormone axis

    Endocrinology, Vol. 154, Núm. 10, pp. 3589-3598

2012

  1. Craniopharyngiomas express embryonic stem cell markers (SOX2, OCT4, KLF4, and SOX9) as pituitary stem cells but do not coexpress RET/GFRA3 receptors

    Journal of Clinical Endocrinology and Metabolism, Vol. 97, Núm. 1

  2. Defining stem cell types: Understanding the therapeutic potential of ESCs, ASCs, and iPS cells

    Journal of Molecular Endocrinology, Vol. 49, Núm. 2

2010

  1. Functional role of the RET dependence receptor, GFRa co-receptors and ligands in the pituitary

    Frontiers of Hormone Research, Vol. 38, pp. 127-138

  2. New insights into thyroglobulin pathophysiology revealed by the study of a family with congenital goiter

    Journal of Clinical Endocrinology and Metabolism, Vol. 95, Núm. 7, pp. 3522-3526

2008

  1. Ghrelin localization in the medulla of rat and human adrenal gland and in pheochromocytomas

    Histology and Histopathology, Vol. 23, Núm. 1, pp. 57-65

2007

  1. Role of ghrelin in reproduction

    Reproduction, Vol. 133, Núm. 3, pp. 531-540

2003

  1. Cellular Distribution and Regulation of Ghrelin Messenger Ribonucleic Acid in the Rat Pituitary Gland

    Endocrinology, Vol. 144, Núm. 11, pp. 5089-5097

2002

  1. Glial-derived neurotropic factor and RET gene expression in normal human anterior pituitary cell types and in pituitary tumors

    Journal of Clinical Endocrinology and Metabolism, Vol. 87, Núm. 4, pp. 1879-1884